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Emerging data reveal that CYP51A1 influences diverse biological processes, including cell proliferation, immune regulation, and multiple forms of regulated cell death such as apoptosis, ferroptosis, alkaliptosis, and potentially pyroptosis
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β-catenin mutations in N-terminal serine/threonine residues or adjacent residues that interrupt CK1- and GSK3β-induced regulatory serine/threonine phosphorylations have been found in several cancers ( Figure 3B )
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